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In conclusion, the gutkidney axis represents a pivotal bidirectional mechanism in CKD progression, wherein gut dysbiosis and uremic toxin overproduction drive a selfperpetuating cycle of inflammation, oxidative stress, immune dysregulation, and renal fibrosis
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* Correspondence: Celia Quijano, [email protected], [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
Furthermore, the correlation between genes in these modules and marker genes of innate immune cells was examined ( Figure 4C )
Molecular Weight: 1024.2 g/mol
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