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during muscle regeneration, it coordinates with matrix metalloproteinases to mobilize precursor cells and remodel the extracellular matrix processes fundamental to bouncing back from hard sessions or setbacks

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It blocks DNA methylation by inhibiting DNA methyltransferase and induces cell cycle arrest in the G2/M phase through the p53-independent pathway in human cancer cells

In targeted therapy, GSH depletion prevents redox-driven adaptive resistance to PI3K/AKT/mTOR, MAPK, and KRAS inhibitors, intensifying oxidative stress and restoring drug sensitivity