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These compositional disruptions contribute to increased intestinal permeability and sustained peripheral inflammation, creating conditions conducive to -synuclein misfolding within the enteric nervous system and its subsequent propagation along gutbrain pathways [25]

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Briefly, 10 6 PFU/mL of each virus (WT and Tamiflu-resistant) was plated in a 96-well plate, and then, various concentrations of OHT were added to each well