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As the prevalence of T2DM increases globally and is recognized as a reemerging risk and challenge to manage TB, more research should investigate the possible benefits of using L-GSH as a potential adjunct therapy alongside current anti-TB treatments or even as a preventative strategy for individuals with T2DM with known dysregulation of GSH metabolism and increased oxidative stress

Proteins are much larger and more complex in structure, consisting of one or more long chains of over 50 amino acids

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G.van der KolkJ.BetsD.et al (2010)

The inflammatory milieu subsequently drives hepatic stellate cell activation and transdifferentiation into myofibroblasts, leading to excessive extracellular matrix deposition and progressive fibrosis, which represents the primary determinant of long-term liver-related outcomes [17]