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HIV-1 Tat-mediated microglial senescence by miRNA-505-SIRT3 pathway Senescence was initially defined as the cessation of cell division after multiple replication cycles [62] and has expanded to include premature senescence triggered by telomere shortening, oxidative stress, mitochondrial dysfunction, and lysosomal alterations [63]
J Pharmacol Exp Ther 1998
Access this chapter Subscribe and save Buy Now Tax calculation will be finalised at checkout Purchases are for personal use only Similar content being viewed by others References Al-Majed AA, Gdo AM, Al-Shabanah OA, Mansour MA (2002) Alpha-lipoic acid ameliorates myocardial toxicity induced by doxorubicin
Zhu, L., et al
Inflammopharmacology 7:114 Lovric-Bencic M, Sikiric P, Hanzevacki JS, Seiwerth S, Rogic D, Kusec V, Aralica G, Konjevoda P, Batelja L, Blagaic AB (2004) Doxorubicine-congestive heart failure-increased big endothelin-1 plasma concentration: reversal by amlodipine, losartan, and gastric pentadecapeptide BPC157 in rat and mouse
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