Diabetes-associated excess kidney tissue RAS activation increases angiotensin II-mediated glomerular efferent arteriolar vasoconstriction and glomerular hyperfiltration, tubular sodium reabsorption, and signalling through intracellular pathways, resulting in inflammation and oxidative stress [13]
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Such a formation of (MeHg) 2 S from MeHg was also seen in the rectal content of another mammal, the small Indian mongoose (Supplemental Fig
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