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Published research has demonstrated that GHK-Cu can influence numerous genes involved in: extracellular matrix synthesis collagen turnover tissue remodelling inflammatory signalling antioxidant defence stem-cell related pathways angiogenesis Copper binding is essential to this biological activity, allowing GHK-Cu to participate in cellular signalling processes associated with tissue maintenance and repair

eGLP-1 showed a markedly lower potency at both human and platypus receptors that was even lower than OXM

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Note: The studies presented in this table describe direct effects of GLP-1 RAs, on macro- and microvascular coronary arteries