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Corticosteroids, IVIG, cyclosporine, TNF- inhibitors, and plasma exchange have some beneficial effects on mitigating skin lesions [26], while the evidence for lowering mortality remains controversial

The resulting cysteine is unstable at extracellular level, and it is rapidly autoxidizes to cystine

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In this study, GSH inhibited ferroptosis by activating the SLC7A11/GPX4 axis and reducing LPO levels to ultimately alleviate graft injury and promote survival