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TNF-, NF-B, IL-1b, AP-1, and Mitogen-activated protein kinases (MAPKs) intermediates get activated after receiving the pro-inflammatory stimuli and release pro-inflammatory chemokines, MMPs, inflammatory enzymes, adhesion molecules, and angiogenic factors resulting in neo-vascularization, arthritic joint destruction and chronic inflammation

We chose the monothiol mutant PfGrx C32S/C88S for our studies to avoid side reactions that complicate the steady-state kinetics 33

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Du Y, Piscitelli D, Ahmad S, Trahair L, Greenfield J, Samocha-Bonet D, Rayner C, Horowitz M, Jones K