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Desmosterol in the cholesterol loading studies was quantified by stable isotope dilution GC/MS analysis

The treatment also decreased pro-inflammatory and pro-fibrotic gene expression with reduced macrophage infiltration (Li et al., 2025)

Conversion of glutathione to glutathione disulfide by cell membrane-bound oxidase activity

Frataxin-knockout mouse myoblasts, heart, and skin fibroblasts all showed characteristics of ferroptosis, and Nrf2 was downregulated in Friedreichs Ataxia patients and frataxin-knockout mice, while Nrf2 activators EPI-743 or sulforaphane regulated redox imbalances and rescued ferroptosis in frataxin knockout cells

5B, C), although the Nrf2 gene was less efficiently deleted compared to the deletion of Gclc