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The GstA1 gene, which encodes a 29 kD GST protein (GST29) was specifically inducible by fungal infections and exogenous GSH, but not by various xenobiotics that typically induce GST activity (paraquat, atrazine, alachlor, metolachlor) (Mauch and Dudler, 1993)

Oxidative stress and heart disease are connected through several biological pathways, including endothelial dysfunction, inflammation, lipid oxidation, and mitochondrial injury

By activating two distinct but complementary pathways, the combination triggers a much more significant and controlled GH release in lab models than either peptide could ever manage on its own

Potential Benefits GH / IGF-1 (theorized): Increased GH and IGF-1 may support tissue repair in certain models

NFB then triggers other cells to release cytokines