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Pre-clinical rodent models of tendon rupture have shown combined BPC-157 and TB-500 protocols to produce faster return of tensile strength and superior histological organisation compared to either single-peptide treatment or untreated controls

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Increased hepatic glucose production contributes to the development of MASLD by driving hyperglycemia and consequent hyperinsulinemia, but hyperglycemia per se (independent of insulin) can also promote liver lipid accumulation by promoting the nuclear translocation of carbohydrate-responsive element binding protein (69)

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