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The CaMK4 signalling pathway has emerged as an important regulator of homoeostatic plasticity, which is a key process by which excitatory and inhibitory signals in neurons are actively balanced to prevent the development of hyper or hypoactivity [104]

Neuroendocrine experiments use pituitary cell cultures (GH3, AtT-20) and primary pituitary tissue explants to measure peptide release in response to DSIP

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