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333 Tryptophan catabolism was found to contribute to the escape of the immune response through accumulated catabolites, creating an immunosuppressive microenvironment in tumors and lymph nodes, with the catabolites binding to and activating AhR

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La forme oxyde, bien moins chre produire, a des effets inverses que la forme rduite: elle est pro-oxydante

Furthermore, pericyte degeneration and apoptosis, as evidenced by the detachment of PDGFR- from pericytes, are induced by accumulated A and impaired A clearance, leading to reduced CBF [45, 74, 146, 180]