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Indeed, selective activation of PGC-1 within the hepatocytes protects the liver from lipids overload and progression to fibrosis, due to its ability to induce mitochondrial oxidative phosphorylation, fatty acids -oxidation and citrate cycle, while decreasing oxidative stress and promoting triglycerides excretion in the bloodstream 15

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Researchers reported changes in collagen-related measurements, including collagen IV

C32 is the second active-site cysteinyl residue of PfGrx and C88 is the third cysteinyl residue, which is in the proximity of the active site and can also undergo redox modifications 14,26,30