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10.1038/s41419-020-02772-8 Cell Death Dis
Consequently, the observed antioxidant effects of the tested agents may be responsible for the depicted hampering in HMGB1 expression by these agents that resulted in the illustrated hindering in RAGE, which dampened the activation of inflammatory signaling, resulting in the detected suppression in the production of pro-inflammatory mediators in these groups
Recent studies link glutathione loss to the progression of neurodegeneration
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