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Additionally, further analyses investigated potential relationships between changes in Glx/tNAA and clinical measure scores

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A secondary effect of this stress is activation of Nrf2 and its target genes resulting in compensatory higher GSH levels and induction of antioxidant genes in the livers of these mice, which makes these livers more resistant to additional acute stress such as APAP toxicity [151]

And if so, what does this mean for the alcohol industry