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In terms of mechanisms, following release, GH binds to its receptor leading to the recruitment and phosphorylation of Janus kinase 2 (JAK2) and its most recognized downstream target, signal transducer and activator of transcription 5 (STAT5) (Jrgensen et al., Figure 1)

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Nonalcoholic fatty liver disease has become one of the hepatic diseases of greatest interest recently due to its epidemiological impact, with impressive clinical and physiopathologic advances and more profound knowledge about its mechanisms that lead to persistent fibrosis, which is intimately related with NRF-2

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