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Furthermore, our in vitro pull-down binding assay showed that the HGF R242E/K244E/R249E mutant indeed exhibited weaker c-MET binding affinity, as compared to wild-type HGF (Fig

Animal studies have shown that GLP-1RAs control appetite and binge eating behavior through serotonin pathways

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Moreover, in facing various pathological challenges, the mammalian heart, which possesses high levels of mitochondria and weak antioxidant capacity, is susceptible to oxidant production and oxidative damage