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It delineates the key regulatory steps of ferroptosis during infections caused by various pathogens, including viruses, bacteria, fungi, and parasites
doi: 10.1038/nri1457 81 PlattenMHoPPYoussefSFontouraPGarrenHHurEMet al
The model also predicted that impairing TCA-dependent NADPH production would elevate the GSSG/GSH ratio and alleviate ER stress, similarly to etomoxir treatment
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[6] As a consequence of its potential in digestive functioning, the peptide was hypothesized to increase appetite and weight gain
Down-stream, the relative loss of mitochondrial function and other knock-on effects, e.g., peroxynitrile-induced toxicity, lead to JNK/Alf-mediated necrosis in the liver (Hinson et al
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