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These new findings encourage a novel working hypothesis for further research, that a chemical-modification mechanism enables transformation of HGF through interaction or reaction with LASSS under physiological conditions, resulting in the retention of HGFs biological activity after exposure to peroxynitrite, at least until an enhanced form of HGF undergoes metabolic turnover in vivo
Role of proinflammatory cytokines in the pathophysiology of osteoarthritis
Cai J, et al
What does glutathione do
Increased endothelial apoptotic cell density in human diabetic erectile tissuecomparison with clinical data
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