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The implication of mitochondrial dysfunction in association with key regulatory factors and signaling pathways involved in the pathogenesis of CKD will be discussed in the context of mitochondrial biogenesis and oxidative capacity, as well as mitochondrial dynamics

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Collectively, these data confirm the functional absence of MZB in Gclc fl/fl Mb1-Cre + mice and demonstrate that Gclc is critical for FoB-mediated GC reactions and antibody-mediated immunity to infections

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