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As a result, transfusion of G6PD-deficient RBCs may be associated with reduced post-transfusion survival, increased hemolysis, and compromised oxygen delivery, particularly in recipients exposed to oxidative stress conditions such as infection or inflammation (71, 76)

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In conclusion, PCs can alleviate diabetes-induced myocardial hypertrophy, cardiac fibrosis, and heart failure, demonstrating potential cardioprotective effects by regulating the S100A11/RAGE/PPAR signaling pathway, improving mitochondrial function, reducing oxidative stress, and inhibiting the formation of AGEs

Hypoxia signaling in human health and diseases: implications and prospects for therapeutics