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The accumulation of free cholesterol in LSEC endolysosomes disrupted Rab7 membrane trafficking and impaired the transport of TLR9 from late endosomes to lysosomes, enhancing activation of the TLR9/inflammasome pathway mediating APAP-induced liver injury

Further validation of its efficacy in clinical applications is needed in the future

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Combined use of nitroglycerin and N-acetylcysteine in the management of unstable angina pectoris

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