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When sleep becomes fragmented or shallow: Pain sensitivity increases Inflammatory signaling rises Tissue repair slows Recovery capacity declines Sleep disruption alone can sustain chronic pain physiology, even in the absence of ongoing structural injury

The pivotal trials enrolled HIV patients with lipodystrophy-associated visceral fat accumulation

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Similarly, total BALF cellularity was elevated in IL-33 challenged mice but notably not in those co-treated with melatonin and Luzindole ( Figure 2C )