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Tumor growth is driven by intricate interactions between the tumor microenvironment (TME) and immune cells, inflammatory mediators, extracellular matrix remodeling, hypoxia, cancer stemness, cellular senescence, and metabolic dysregulation

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Rather than positioning glutathione as a single-action compound, the webinar framed it as part of a broader intracellular control network that influences how cells respond to oxidative stress and metabolic demand