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First, the human RCT footprint is thinner than the mechanism literature

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Taken together, these data suggest that vaspin exerts multiple mechanisms to control adrenergic activation of brown adipocyte metabolism and lipid turnover through interactions with the LDLR family, thereby inhibiting cAMP-dependent neutral lipolysis via modulation of PDE activities and blocking endocytotic lipid trafficking towards lysosomal acid lipolysis

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This contributes to impaired FSH signaling and increased production of androgens