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The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

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Geoghegan J

When research on a particular compound comes almost exclusively from one geographic region and institutional network, there is inherent risk of confirmation bias, methodological preferences that favor positive findings, and results that may not generalize to other populations or research contexts