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Melanotan I, being more selective for MC1R, remains more skin focused in its pharmacology and is typically administered as a slow-release subcutaneous implant in its approved medical form, smoothing out plasma concentrations and reducing peaks that can amplify side effects

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By binding glutamate, NMDA receptors (NMDAR) gate an influx of Ca 2+ and Na + to cause membrane depolarization, while GABA binds to GABA A receptors, an influx of Cl results in hyperpolarization [5]