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Exploring the crosstalk among these pathways and the regulatory mechanisms that govern them is critical for developing therapeutic strategies

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45 Interestingly, while UBE1 expression remains unchanged in human heart failure, experimental studies have shown that overexpression of UBE1 or UBE6 in neonatal rat ventricular cardiomyocytes (NRVCMs) increases the ubiquitination of Nav1.5, a major transmembrane protein essential for cardiomyocyte depolarization

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Consequently, optimizing treatment to prevent refractory infection and exploring microbiota-modulating strategies are highlighted as crucial steps in management (Li et al., 2025)