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Multiple mechanisms are proposed in the onset and progression of neuro-COVID including hypoxia, hyper-coagulability, endothelial dysfunction, nerve injury, neuro-inflammation, and neurotropism, where all conditions are induced by SARS-CoV-2 infection

Panickar et al., 2009), especially on the motor neurons (Koh et al., 2006) because it crosses the blood-brain barrier and modulates mitochondrial responses to OS (Table 1)

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