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The resulting holoenzyme dimer comprising the two subunits is catalytically more efficient and less subject to inhibition by glutathione (GSH) than GCLC alone

Magnesium and B-Complex address ATP synthesis from two distinct biochemical pathways simultaneously, one as a mineral cofactor and the other through enzymatic coenzyme activity

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DPT3f enhances FSP1/CoQ 10 pathway antioxidant activity by upregulating FSP1 protein expression, promotes CoQ 10 conversion to reduced CoQ 10 H 2 to scavenge lipid peroxides, thus inhibiting dopaminergic neuron ferroptosis, protecting SN neurons and alleviating PD-related damage (230)