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In this review, we examine GLP-1RA therapy from a metabolism-centered perspective, integrating incretin signaling with substrate redistribution, mitochondrial metabolic throughput, and tissue-specific energetic constraints

10.1007/s11010-019-03588-7 20 ChengJ.WuL

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Moreover, chronic oxidative stress generated through CYP2E1 induction was shown to interfere with the insulin signaling cascade via a decrease in tyrosine phosphorylation of insulin receptor substrates 1 and 2