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A Clinical Guideline

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Interestingly, a role for UCP2 in the transport of mGSH has been described in neurons, suggesting that the transport of protons back into the matrix by UCP2 may favor the movement of GSH (de Bilbao et al., 2004)

No obvious histological damages could be observed in the saline group, whereas, the tumor slice of DOX/LAAM GSH-CQD-treated mice exhibited nuclear shrinkage and disappearance area, indicating a higher treatment effect on tumors