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A possible reason for this is that there is a higher background concentration of carnitine in muscle than in the liver, or that the degree of supplementation with l- and d-carnitine was insufficient to significantly alter the concentration of acyl-carnitine, such that any change in lipid metabolism in muscle was not obvious
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Thaci B, Brown CE, Binello E et al (2014) Significance of interleukin-13 receptor alpha 2-targeted glioblastoma therapy
[PubMed] [Google Scholar] 44.Churchward-Venne T.A., Cotie L.M., MacDonald M.J., Mitchell C.J., Prior T., Baker S.K., Phillips S.M
QXJYG reduced inflammatory factors associated with ferroptosis, lowered lipid peroxides such as MDA, enhanced antioxidant capacity, and inhibited AS progression and plaque vulnerability
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