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The body's master antioxidant in a vegan-friendly, natural, high-bioavailability formula

Sturm, A

Although additional studies are required to clarify the molecular mechanisms responsible for hyperhomocysteinemia-induced vascular disorders, there is evidence that the direct cytotoxicity of markedly elevated homocysteine and/or of its oxidized by-products (homocysteine thiolactone, homocysteine sulfinic acid, and homocysteic acid) damages endothelial cells (Dudman et al, 1991

To a lesser extent, CYP1A1 also participates in drug metabolism, thus co-administering CYP1A1 inducers can decrease bedaquiline concentrations neglecting its effectiveness

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