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Glucose metabolism has been shown to modulate the effector function of T H 17 cells by controlling a metabolic-transcriptional network, with Acly serving as a key rate-limiting mediator generating acetyl-CoA and dictating T H 17 cell pathogenicity 21 , while restraining the development of FOXP3 + T regs 74

The following primers were used: AgRP (forward: ctttggcggaggtgctagat

Lu K, Sun J, Li Q, Li X, Jin S

References & More Injection Techniques in Musculoskeletal Medicine