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Oriented endocytic recycling of 51 in motile neutrophils

Thus, the transition from "physiological" to "pathological" fibrosis is driven by impaired apoptosis of myofibroblasts, as well as excessive ECM deposition and accumulation, resembling a persistent wound-healing response

The net effect of ERR activation is increased mitochondrial mass and function, enhanced oxidative capacity, greater fatty acid utilization, and improved metabolic flexibility

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