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I like it best, the way Lenzi A et al said it in their paper Rationale for Glutathione Therapy published in the Oxford journal Human Reproduction

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Zn-deficient wild-type and mutant human SOD1 have been implicated in the disease familial amyotrophic lateral sclerosis (FALS), also known as Lou Gehrigs disease which affects the nerve cells in the spinal cord and the brain.29, 30 Recently, Dayal and colleagues informed that deficiency of superoxide dismutase promoted cerebral vascular hypertrophy and vascular dysfunction in hyperhomocysteinemia.31 SOD enzyme deficiency is common

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