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Their results demonstrate that GLP-1, through an unknown mechanism, can preserve axonal and neurofilament structure despite the induction of progressive neurotoxicity, thus illustrating neuroprotective properties that can be leveraged against disorders in the central nervous system [60]

These effects could be attenuated by lowering TMAO levels or pharmacological inhibition of Smad3 signaling (Li et al., 2019a)

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Zheng et al., 2015, 2016)

Moreover, following systemic administration, mGIPR-Ab has access to the brain and results in weight loss comparable to central administration of mGIPR-Ab