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These proinflammatory molecules also induce the above lesions through the nuclear factor-B (NF-B), Janus kinase/signal transducers and activators of transcription (JAK/STAT), and TGF-/Smad signaling pathways[99-101]

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Cascella R, Evangelisti E, Zampagni M, Becatti M, DAdamio G, Goti A, Liguri G, Fiorillo C, Cecchi C