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Putative mechanisms of action include: Decreased production of pro-inflammatory cytokines: curcumin can block the production of IL-12 by CD4, CD8, and NK cells, thereby reducing inflammation [77]

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Furthermore, therapeutic mechanisms such as alternative splicing ASOs that have FDA approval for non-NDD pathologies such as Spinraza (SMA) and Eteplirsen (DMD) may provide a strong basis for further studies using these ASOs for genes with splice mutations implicated in NDDs

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