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In a rat model of IBS, intraperitoneal administration of exendin-4 normalized stress-induced defecation and visceral pain sensitivity by modulating enteric neuronal function through GLP-1 receptors in submucosal and myenteric ganglion neurons [52]

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[94] A preliminary study with human subjects indicated they did produce the class of T-cells required (in theory, at least) to suppress autoimmunity in the brain, but with appreciable subject-to-subject variation