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PI3K activates protein kinase B (Akt), which inactivates several important substrate proteins, such as glycogen synthase kinase 3 (GSK-3) 18 and forkhead box O, 19 and activates mammalian target of rapamycin (mTOR)

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Emotional eating, binge-eating tendencies, psychiatric comorbidity, learned eating behaviour, reward sensitivity, and baseline dietary patterns may all shape how patients perceive and respond to GLP-1RA-induced changes in satiety, nausea, food preference, and hedonic evaluation