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The blunted weight loss in mGIPR-Ab/P1-treated Glp1r Wnt1/ mice was associated with less reduction in food intake compared to Glp1r fl/fl controls (Fig

Modulation of mitochondrial membrane potential and ROS generation by nicotinamide in a manner independent of SIRT1 and mitophagy

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Table 1 ), indicating that yeast endogenous GGT could not use -glutamyl- S -allyl-L-cysteine as a -glutamyl donor substrate

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