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Interestingly, cladribine is cytotoxic not only to dividing cells, but also to resting cells, suggesting alternative mechanism of action in non-dividing cells such as caspase- dependent (136) and caspase-independent (137) mechanisms, via direct mitochondrial toxicity (137, 138), inhibition of DNA repair (139) and epigenetic alterations (140)

The antioxidative glutathione pathway is found to be central to metabolic integration and reprogramming in inflammatory responses mediated by T-cells

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