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In AD mouse models, pathophysiological changes, such as anti-apoptotic effects, reduced A deposition and phosphorylated hippocampal tau, protection from synapse loss, reduced inflammatory and oxidative stress response in the cortex and hippocampus, and increased BDNF have been observed [119,120,121]

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That's based on research involving rats, however, and no studies done on humans have established the same connection

Zinc can activate the Nrf2/HO-1 pathway, increase the contents of GPX4, SOD and GSH, reduce the levels of lipid peroxides, MDA and ROS, and effectively reverse the behavioral and structural changes after SCI