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The importance of cellular redox balance on ciliary function is further illustrated in the case of respiratory syncytial virus (RSV) infections, which the imbalanced ROS production in the airway epithelial microenvironment not only leads to oxidative damage to cilia but also disrupt the function of several redox-sensitive proteins such as protein kinase A (PKA), PKC, and protein phosphatase 1, which play crucial roles in regulating ciliary motility, eventually exacerbating airway mucociliary dysfunction (65, 8083)