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Catalpol not only inhibits the over-proliferation of astrocytes in the ischemic cortex and DG, preventing neuroinflammation and scar formation from affecting the migration of newborn neurons, but also promotes recovery in the ischemic brain area by enhancing activation, providing neurotrophic substances, and supporting neuron survival, thus creating a favorable microenvironment for the migration of newborn neurons to the ischemic cortex area (Sun, 2022

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