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PUFAs add fuel to Crohns disease-associated AIEC-induced enteritis by exacerbating intestinal epithelial lipid peroxidation
Serrapeptase, nattokinase, N-acetylcysteine (NAC), and EDTA-based formulations degrade the biofilm scaffold, exposing the embedded yeast cells to antifungal agents that would otherwise be ineffective
Distinguishing between these requires controlled conditions and objective markers, neither of which most patients track
This quenching of the initiating perferryl radicals, which prevent propagation of lipid peroxidation, protects not only lipids but also proteins from oxidation
CAMP activates p38 MAPK, phosphorylates CREB and promotes MITF expression (Kang et al., 2020
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